The inferior turbinates are long, narrow structures along the side walls of the nose. They play a crucial role in humidifying, warming, and filtering the air we breathe. They also contain airflow receptors, which signal the sensation of air entry to the brain, and their torpedo shape helps direct a smooth, laminar stream of air from the front to the back of the nose.
When enlarged, however, the turbinates can obstruct airflow — particularly at night or during physical exertion — leading to persistent nasal congestion.
Despite the common label of “inferior turbinate hypertrophy,” this term is often inaccurate. In most cases, the issue is vascular engorgement, not permanent overgrowth. The distinction is critical to appropriate diagnosis and treatment.
Mr Patel recognises that most cases of turbinate enlargement are not caused by structural hypertrophy but by tissue engorgement — a dynamic, reversible process driven by vascular dilation.
This explains why many patients experience immediate relief with vasoconstrictors (e.g. xylometazoline). True hypertrophy would not resolve so quickly.
Understanding this difference is central to Mr Patel’s approach. He uses structured, physiology-first testing to determine whether the problem is mucosal, structural, or mixed — and tailors treatment accordingly.
Mr Patel performs a detailed same-day assessment, including:
Medical
Surgical
There are several techniques available to reduce turbinate size. These include:
Mr Patel selectively offers only those techniques that align with his function-preserving philosophy.
Not all enlarged turbinates need surgery. Mr Patel’s priority is to identify whether the enlargement is reversible mucosal engorgement or bony hypertrophy, and to address the underlying cause — not just the symptoms.
He aims to preserve normal turbinate function, including its shape, sensory receptors, and surface area, avoiding techniques that remove or destroy these critical structures.
Where surgical reduction is required, Mr Patel prefers PLNA ligation, which addresses the underlying vascular cause by reducing blood flow to the turbinate. For patients undergoing concurrent procedures (such as septoplasty), he may offer more limited techniques where appropriate.
All patients undergoing PLNA Ligation surgery under Mr Patel’s care are monitored using both objective airflow testing and patient-reported outcome measures. This allows for transparent, data-driven evaluation of results.
Nasal Airflow Improvement
Patients experience an average 48% increase in nasal airflow at 3 months following PLNA Ligation, as measured by Nasal Inspiratory Peak Flow (NIPF) testing.
Note: All measurements are taken without nasal decongestants to reflect natural breathing capacity.
Symptom Reduction
On average, patients report a 91% reduction in nasal obstruction symptoms at 3 months post-operatively, based on NOSE scores.
Safety Data
No patients have experienced a post operate haemorrhage following PLNA Ligation. Post operative haemorrhage can be seen more frequently with some more aggressive techniques for turbinate reduction
Copyright © 2025 London ENT Surgery - All Rights Reserved.